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Paxil

paroxetine

SSRIFDA 1992
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Boxed Warning

Suicidality risk in children, adolescents, and young adults under 25 during initial treatment.

Overview

Paroxetine is an SSRI with notable anticholinergic activity, approved for major depressive disorder, OCD, panic disorder, social anxiety disorder, GAD, and PTSD. It is widely recognized as one of the more difficult SSRIs to discontinue.

Common Doses

10mg, 20mg, 30mg, 40mg

Formulations

Tablets: 10mg, 20mg, 30mg, 40mg; Oral suspension: 10mg/5mL; Controlled-release (CR) tablets: 12.5mg, 25mg, 37.5mg

Pregnancy

Category D (positive evidence of risk — cardiac malformations)

Mechanism of Action

Potent and selective inhibitor of serotonin reuptake (SERT). Also has significant anticholinergic (muscarinic) activity and weak norepinephrine reuptake inhibition, which contribute to its withdrawal profile.

Taper Notes

Short half-life for an SSRI. Liquid formulation available. Often considered one of the harder SSRIs to taper.

Maudsley Deprescribing Guidance

Particularly difficult to stop due to short half-life and anticholinergic withdrawal. Very slow final taper essential.

Common Withdrawal Symptoms

brain zapsdizzinesselectric shockscrying spellsflu-like symptoms

Interactions & Safety

Drug Interactions

  • MAOIs — contraindicated (serotonin syndrome risk)
  • Thioridazine — contraindicated (QT prolongation via CYP2D6 inhibition)
  • Pimozide — contraindicated

Food Interactions

  • No significant food effect on absorption
  • Avoid alcohol during treatment

Contraindications

  • MAOIs within 14 days
  • Thioridazine
  • Pimozide

Toxicity

Serotonin syndrome risk. Anticholinergic toxicity at high doses. Associated with higher rates of discontinuation syndrome than other SSRIs.

Pharmacokinetics

ADME Profile

Absorption

Completely absorbed after oral dosing but extensive first-pass metabolism reduces bioavailability to ~50%. Tmax ~5 hours. Food does not significantly affect absorption.

Distribution

~8.7 L/kg

Metabolism

Extensively metabolized hepatically via CYP2D6 (primary) with CYP3A4 contribution. Paroxetine inhibits its own metabolism (CYP2D6 saturation), leading to non-linear pharmacokinetics.

Elimination

Renal (~64% as metabolites, ~2% unchanged) and fecal (~36%).

Protein Binding

~93–95%

Clearance

Non-linear; clearance decreases at higher doses due to CYP2D6 saturation.

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